2022年2月5日土曜日

自然免疫を狂わすm1Ψ (N1-Methylpseudouridine)が齎らすちょっとした事で簡単に砕け散るガラスの肉体



 N1-Methylpseudouridine (abbreviated m1Ψ) is a natural archaeal tRNA component[1] as well as a synthetic pyrimidine nucleoside used in biochemistry and molecular biology for in vitro transcription and is found in the SARS-CoV-2 mRNA vaccines tozinameran (PfizerBioNTech) and elasomeran (Moderna).[2]: 1 It had previously been tested in vaccines against Zika,[3][4][5] HIV-1,[5] influenza,[5] and Ebola[6] in 2017–2018.[2]: 5

Properties

N1-Methylpseudouridine is the methylated derivative of pseudouridine. It is used in in vitro transcription and for the production of RNA vaccines,[7][8] In vertebrates, it stimulates significantly less activation of the innate immune response compared to uridine.[9] At the same time, the translation is stronger.[10][11] In protein biosynthesis, it is read like uridine and enables comparatively high protein yields.[11][12] In 2016, a protocol for large-scale synthesis of the nucleoside triphosphate from the ribonucleoside was published.[13] The nucleoside itself can be made by chemical methylation of pseudouridine.[14]

While pseudouridine can wobble-pair with bases other than A,[15] potentially leading to mistranslated proteins, it is unclear whether this happens with m1Ψ.[16][2]: 4[contradictory]

 


 The innate, or nonspecific, immune system [1]is one of the two main immunity strategies (the other being the adaptive immune system) in vertebrates. The innate immune system is an older evolutionary defense strategy, relatively speaking, and is the dominant immune system response found in plants, fungi, insects, and primitive multicellular organisms (see Beyond vertebrates).[2]

The major functions of the innate immune system are to:

  • recruit immune cells to infection sites by producing chemical factors, including chemical mediators called cytokines
  • activate the complement cascade to identify bacteria, activate cells, and promote clearance of antibody complexes or dead cells
  • identify and remove foreign substances present in organs, tissues, blood and lymph, by specialized white blood cells
  • activate the adaptive immune system through antigen presentation
  • act as a physical and chemical barrier to infectious agents; via physical measures such as skin and chemical measures such as clotting factors in blood, which are released following a contusion or other injury that breaks through the first-line physical barrier (not to be confused with a second-line physical or chemical barrier, such as the blood-brain barrier, which protects the nervous system from pathogens that have already gained access to the host).


Inflammation is one of the first responses of the immune system to infection or irritation. Inflammation is stimulated by chemical factors released by injured cells. It establishes a physical barrier against the spread of infection and promotes healing of any damaged tissue following pathogen clearance.[5]

The process of acute inflammation is initiated by cells already present in all tissues, mainly resident macrophages, dendritic cells, histiocytes, Kupffer cells, and mast cells. These cells present receptors contained on the surface or within the cell, named pattern recognition receptors (PRRs), which recognize molecules that are broadly shared by pathogens but distinguishable from host molecules, collectively referred to as pathogen-associated molecular patterns (PAMPs). At the onset of an infection, burn, or other injuries, these cells undergo activation (one of their PRRs recognizes a PAMP) and release inflammatory mediators responsible for the clinical signs of inflammation.

Chemical factors produced during inflammation (histamine, bradykinin, serotonin, leukotrienes, and prostaglandins) sensitize pain receptors, cause local vasodilation of the blood vessels, and attract phagocytes, especially neutrophils.[5] Neutrophils then trigger other parts of the immune system by releasing factors that summon additional leukocytes and lymphocytes. Cytokines produced by macrophages and other cells of the innate immune system mediate the inflammatory response. These cytokines include TNF, HMGB1, and IL-1.[6]

The inflammatory response is characterized by the following symptoms:




血液凝固コントロールの重要な役割を補ってる自然免疫が狂うとどなるか。
炎症反応がまったく起きない身体になるとどうなるか。
Death汁接種者の皆様は一度よおく考えてみませう。(爆wwwwwwwwww


2021/10/10 — シュードウリジン (英: pseudouridine、ギリシャ文字のΨや英文字のQで略記され ... RNAに知られている100を超える修飾塩基としては最も一般的で、Ψは ...
2021/12/26 — 2021年10月10日日曜日. mRNAワクチンを可能にした「ヤマサ醤油」の「シュードウリジン」がΨ難の素かもしれない件.



2021年12月26日日曜日

日本の醤油屋の技術で身体が「毒」と認識しないので人体の修復機能が戻してくれる可能性は低いと思われ




2021年11月25日木曜日

「シャブ中」の「シャブ抜き」は可能だが 「mRNAジャブ中」の「mRNAジャブ抜き」はほぼ不可能


2021年10月10日日曜日

mRNAワクチンを可能にした「ヤマサ醤油」の「シュードウリジン」がΨ難の素かもしれない件




Ψ can Trick the Immune SystemThis landmark study indicated the importance of this RNA modification in mRNA platform technologies. Ψ can Trick the Immune ...




Ψ


site://tokumei10.blogspot.com Innate

site://tokumei10.blogspot.com Innate

site://tokumei10.blogspot.com Innate


Dr. Ryan Cole has called the COVID-19 vaccine a "clot shot," "needle rape," an "experimental" drug and a "poisonous attack on our population" that must be stopped.


、、、(爆wwwwwwwwwwww

3 件のコメント:

匿名 さんのコメント...

> N1-Methylpseudouridine (abbreviated m1Ψ)

M1-Ψ難グランプリwwwwwwwwwwwwwwwwwwwwwwwwwwwwwwwwwwwwwwwwww
uridine・・・
ウリジーン・・・
ウリ Gene wwwwwwwwwwwwwwwwwwwwwwwwwwwwwwwwwwwwwwwwwwwww

匿名 さんのコメント...

吠えない番犬

猿轡ならぬ犬轡

やっぱヤマサ(爆

GABRIEL さんのコメント...

易感染と出血



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